ArticleMolecular medicine reports2026
NF‑κB signaling drives Th17‑mediated lacrimal gland injury and tear dysfunction in a murine model of primary Sjögren's syndrome.
Article in Molecular medicine reports, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
Primary Sjögren's syndrome (pSS) is a systemic autoimmune disorder characterized by chronic inflammation of exocrine glands, resulting in lacrimal gland dysfunction, reduced tear secretion and dry eye manifestations. The nuclear factor κB (NF‑κB) signaling pathway is a central regulator of inflammatory responses; however, its role in lacrimal gland injury in pSS remains incompletely defined. The present study investigated the contribution of NF‑κB signaling to lacrimal gland damage and tear secretion in non‑obese diabetic (NOD)/Ltj mice, an established
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