Article in Nature communications, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registry
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what money
Authors and funding
13 authors.
Jan MeyerInstitute of Neurobiology, Faculty of Mathematics and Natural Sciences, Heinrich Heine University Düsseldorf, Düsseldorf, Germany.ORCID 0000-0002-3351-8653
Viola Bornemann *Institute of Neurobiology, Faculty of Mathematics and Natural Sciences, Heinrich Heine University Düsseldorf, Düsseldorf, Germany.ORCID 0009-0001-5199-7360
Alok Bhattarai *Department of Physics, University of South Florida, Tampa, FL, USA.
Sara EitelmannInstitute of Neurobiology, Faculty of Mathematics and Natural Sciences, Heinrich Heine University Düsseldorf, Düsseldorf, Germany.
Petr UnichenkoInstitute of Cellular Neurosciences I, Medical Faculty, University of Bonn, Bonn, Germany.
Simone DurryInstitute of Neurobiology, Faculty of Mathematics and Natural Sciences, Heinrich Heine University Düsseldorf, Düsseldorf, Germany.
Karl W KafitzInstitute of Neurobiology, Faculty of Mathematics and Natural Sciences, Heinrich Heine University Düsseldorf, Düsseldorf, Germany.
Nicholas ChalmersDepartment of Neurosurgery, University Hospital Erlangen, Friedrich-Alexander-Universität, Erlangen, Germany.ORCID 0009-0004-0778-6840
Jianfeng FanInstitute of Neurobiology, Faculty of Mathematics and Natural Sciences, Heinrich Heine University Düsseldorf, Düsseldorf, Germany.ORCID 0000-0003-2437-3592
Ruth BeckervordersandforthDepartment of Neurosurgery, University Hospital Erlangen, Friedrich-Alexander-Universität, Erlangen, Germany.ORCID 0000-0002-3617-7567
Christian HennebergerInstitute of Cellular Neurosciences I, Medical Faculty, University of Bonn, Bonn, Germany.ORCID 0000-0002-5391-7387
Ghanim UllahDepartment of Physics, University of South Florida, Tampa, FL, USA.ORCID 0000-0002-3716-8742
Christine R RoseInstitute of Neurobiology, Faculty of Mathematics and Natural Sciences, Heinrich Heine University Düsseldorf, Düsseldorf, Germany. rose@hhu.de.ORCID 0000-0002-9684-3592
Funding
CRCNS US-German Research Proposal: Quantitative and Computational Dissection of Glutamatergic Crosstalk at Tripartite SynapsesR01NS130916 · NINDS · UNIVERSITY OF SOUTH FLORIDA · PI Ghanim Ullah · 2023 to 2026
$487k
Investigating the aggregation of amyloid β during metabolic stress using multiscale modelingR21AG087910 · NIA · UNIVERSITY OF SOUTH FLORIDA · PI MUSCHOL, MARTIN, ULLAH, GHANIM · 2024 to 2025
$394k
Bundesministerium für Bildung und Forschung (Federal Ministry of Education and Research) Project SynGluCrossChina Scholarship Council (CSC) 202306370039Deutsche Forschungsgemeinschaft (German Research Foundation) Project #461542557Deutsche Forschungsgemeinschaft (German Research Foundation) RO 2327/13-2,14-2Foundation for the National Institutes of Health (Foundation for the National Institutes of Health, Inc.) R01R01NS130916; R21AG087910NIA NIH HHS R21 AG087910NINDS NIH HHS R01 NS130916
6 · The paper itself
Abstract
Astrocytes maintain extracellular ion and transmitter homeostasis, with the Na⁺ inward gradient playing a crucial role. Earlier studies suggested a rather low, uniform Na⁺ distribution in astrocytes, consistent with the view that these basic homeostatic properties are well-protected. Here, we employed multi-photon fluorescence lifetime imaging to quantitatively determine astrocytic [Na
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.
Cellular and subcellular heterogeneity of astrocytic Na⁺ homeostasis tuning astrocytes into functionally distinct subgroups in the mouse brain. · full record | OpenQuestion