Evidence map›Paper›PMID 42157158›Full record

ArticleBMC biology2026

Integrative interpretable learning reveals shared patterns of epitranscriptomic regulation across multiple cancer types.

Xiangyu Yin, Gang Tu, Xuan Wang, Yuqi Liu, Yue Wang, Jiongming Ma, XiaoXuan Yu, Jia Meng, Bowen Song

Abstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in BMC biology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors.

Xiangyu Yin *Department of Public Health, Department of Pharmacology, School of Medicine, Nanjing University of Chinese Medicine, Nanjing, 210023, China.
Gang Tu *Department of Biosciences and Bioinformatics, Center for Intelligent RNA Therapeutics, Suzhou Key Laboratory of Cancer Biology and Chronic Disease, School of Science, XJTLU Entrepreneur College, Xi'an Jiaotong-Liverpool University, Suzhou, 215123, China.
Xuan Wang *Department of Biosciences and Bioinformatics, Center for Intelligent RNA Therapeutics, Suzhou Key Laboratory of Cancer Biology and Chronic Disease, School of Science, XJTLU Entrepreneur College, Xi'an Jiaotong-Liverpool University, Suzhou, 215123, China.
Yuqi LiuDepartment of Public Health, Department of Pharmacology, School of Medicine, Nanjing University of Chinese Medicine, Nanjing, 210023, China.
Yue WangJiangsu Key Laboratory for Functional Substance of Chinese Medicine, School of Pharmacy, Nanjing University of Chinese Medicine, Nanjing, 210023, China.
Jiongming MaDepartment of Biosciences and Bioinformatics, Center for Intelligent RNA Therapeutics, Suzhou Key Laboratory of Cancer Biology and Chronic Disease, School of Science, XJTLU Entrepreneur College, Xi'an Jiaotong-Liverpool University, Suzhou, 215123, China.
XiaoXuan YuDepartment of Public Health, Department of Pharmacology, School of Medicine, Nanjing University of Chinese Medicine, Nanjing, 210023, China. xxyu@njucm.edu.cn.
Jia MengInstitute of Biomedical Research, Regulatory Mechanism and Targeted Therapy for Liver Cancer Shiyan Key Laboratory, Hubei Provincial Clinical Research Center for Precise Diagnosis and Treatment of Liver Cancer, Taihe Hospital, Hubei University of Medicine, Shiyan, 442000, China. jia.meng@xjtlu.edu.cn.
Bowen SongDepartment of Public Health, Department of Pharmacology, School of Medicine, Nanjing University of Chinese Medicine, Nanjing, 210023, China. bowen.song@njucm.edu.cn.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundCancer is a complex set of diseases caused by the dysregulation of cell proliferation, differentiation, and apoptosis, ultimately leading to malignant tumor development and metastasis. Recent studies have revealed that dysregulation of N4-acetylcytidine (ac

resultsIn this study, leveraging an extensive collection of 88 ac

conclusionsTaken together, our findings highlight the importance of a comprehensive characterization of ac

Indexed as

CytidineGene Expression Regulation, NeoplasticNeoplasmsEpitranscriptomeEpitranscriptomicsHumansCytidineEpitranscriptomic regulationGenomic featuresInterpretable analysisN4-acetylcytidine (ac4C)

Identifiers

PMID42157158
PMCPMC13352858

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.