Evidence map›Paper›PMID 42156946›Full record

ArticleCommunications biology2026

Site-specific DNA double-strand break induces local transcription in cis and protein expression.

Alessia di Lillo, Sara Tavella, Fabio Iannelli, Giovanni Crisafulli, Ubaldo Gioia, Lucrezia A Trastus, Matteo Cabrini, Fabrizio d'Adda di Fagagna

Abstract read
In one paragraph

Article in Communications biology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Alessia di Lillo *IFOM ETS-The AIRC Institute of Molecular Oncology, Milan, Italy.
Sara Tavella *IFOM ETS-The AIRC Institute of Molecular Oncology, Milan, Italy.
Fabio IannelliIFOM ETS-The AIRC Institute of Molecular Oncology, Milan, Italy.
Giovanni CrisafulliIFOM ETS-The AIRC Institute of Molecular Oncology, Milan, Italy.
Ubaldo GioiaIFOM ETS-The AIRC Institute of Molecular Oncology, Milan, Italy.
Lucrezia A TrastusIFOM ETS-The AIRC Institute of Molecular Oncology, Milan, Italy.ORCID 0000-0002-2280-3854
Matteo CabriniIFOM ETS-The AIRC Institute of Molecular Oncology, Milan, Italy.
Fabrizio d'Adda di FagagnaIFOM ETS-The AIRC Institute of Molecular Oncology, Milan, Italy. fabrizio.dadda@ifom.eu.ORCID 0000-0002-9603-5966

Funding

Associazione Italiana per la Ricerca sul Cancro (Italian Association for Cancer Research) 21762Associazione Italiana per la Ricerca sul Cancro (Italian Association for Cancer Research) 22458Associazione Italiana per la Ricerca sul Cancro (Italian Association for Cancer Research) 30471
6 · The paper itself

Abstract

The DNA damage response is a complex network of pathways that cells activate to safeguard genome integrity following DNA damage, including DNA double-strand breaks. We and others previously reported that RNA polymerase II, together with components of the preinitiation complex, is recruited to exposed DNA ends. This results in the assembly of a fully competent transcriptional apparatus and the synthesis of damage-induced long non-coding RNAs, which are necessary for full DNA damage response activation. Thus, DNA double-strand breaks could act as transcriptional promoters. Whether such DNA breaks, generated upstream of an open reading frame lacking a transcriptional promoter and followed by a polyadenylation signal, can induce the transcription of a coding RNA that is subsequently translated into a protein product remains unknown. Here, taking advantage of the CRISPR/Cas9 technology, we generate a sequence-specific double-strand break upstream of a promoter-less, and therefore silent, reporter gene in two distinct cellular systems. In both cell models, a DNA double-strand break is sufficient to trigger the expression of polyadenylated transcripts and a protein product. Collectively, our results demonstrate that DNA double-strand breaks can act as functional promoters capable of driving protein synthesis, revealing an additional mechanism through which DNA damage can regulate gene expression.

Indexed as

DNA Breaks, Double-StrandedTranscription, GeneticCRISPR-Cas SystemsHumansPromoter Regions, Genetic

Identifiers

PMID42156946
PMCPMC13389459

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.