Evidence map›Paper›PMID 42138981›Full record

ArticleMicrobiology (Reading, England)2026

Mutations that confer resistance to sodium azide affect the levels of two physiological isoforms of SecA in

Chen Jiang, Mathew Milner, Georgia Williams, Osama Abdullah, Peter Lund, Damon Huber

Abstract read
In one paragraph

Article in Microbiology (Reading, England), 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Chen JiangSchool of Biosciences, University of Birmingham, Edgbaston, Birmingham, UK.
Mathew MilnerSchool of Biosciences, University of Birmingham, Edgbaston, Birmingham, UK.
Georgia WilliamsSchool of Biosciences, University of Birmingham, Edgbaston, Birmingham, UK.
Osama AbdullahSchool of Biosciences, University of Birmingham, Edgbaston, Birmingham, UK.
Peter LundSchool of Biosciences, University of Birmingham, Edgbaston, Birmingham, UK.
Damon HuberSchool of Biosciences, University of Birmingham, Edgbaston, Birmingham, UK.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Sodium azide inhibits bacterial growth by inhibiting the SecA-dependent translocation of proteins across the cytoplasmic membrane, and all of the mutations known to confer increased resistance to azide are in the

Indexed as

Adenosine TriphosphatasesBacterial ProteinsDrug Resistance, BacterialEscherichia coliEscherichia coli ProteinsMembrane Transport ProteinsMutationSodium AzideMutagenesis, InsertionalProtein IsoformsSecA ProteinsSEC Translocation ChannelsAdenosine TriphosphatasesBacterial ProteinsEscherichia coli ProteinsMembrane Transport ProteinsProtein IsoformsSecA protein, E coliSecA ProteinsSEC Translocation ChannelsSodium AzideazideC-terminal tailprotein translocationSecSecATraDIS

Identifiers

PMID42138981
PMCPMC13178939

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.