ArticleVeterinary research2026
Comprehensive molecular profiling of the African swine fever virus in Korean wild boars between 2019 and 2024.
Article in Veterinary research, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
8 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
African swine fever (ASF), caused by the African swine fever virus (ASFV), is a lethal transboundary disease. Since its first detection in South Korea in 2019, ASFV has continuously infected wild boars, imposing major economic and ecological burdens. We investigated ASFV genetic diversity and transmission dynamics by analyzing 4,209 ASFV-positive wild boar samples collected between 2019 and 2024 using a multi-marker approach, including B646L (p72), the intergenic region (IGR) between I73R/I329L, MGF 360-1La, and MGF 505-9R/10R. All isolates were classified as Genotype II by p72 analysis. IGR II was predominant, while one case of IGR I and three of IGR III were detected in 2019-2020. IGR III reappeared in 2023 and increased in 2024. The Korean-specific MGF 360-1La mutation (L106P) emerged in 2020, peaked in 2021, and persisted thereafter at a low frequency. A shift in the MGF 505-9R/10R profile was observed: MGF-1 dominated until 2020, whereas MGF-5 emerged in 2021 and became predominant in 2024. Accordingly, ASFV strains were classified into six clusters with distinct spatial distributions and transmission trajectories. Cluster 1 represented the initial incursion and early spread, and two minor subclusters (1.1 and 1.2) were detected early on; Cluster 2 remained localized, Cluster 3 expanded southeastward and is emerging as dominant, and Cluster 4 was confined to eastern Gyeongsangbuk-do. These findings suggest that although ASFV in Korea originated from a single Genotype II introduction, the detection of multiple lineages reflects viral diversification or additional incursions. Continuous molecular surveillance using genetic markers, complemented by whole-genome analyses, is essential for detecting early variants and developing effective ASF control strategies.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.