Evidence map›Paper›PMID 42129631›Full record

ArticleCellular & molecular biology letters2026

H

Zhen Ning Tony He, Fangzhou Meng, Xiaoyang Qian, Yuying Liu, Xin Fang, Dan Yang, Qing Yang

Abstract read
In one paragraph

Article in Cellular & molecular biology letters, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Zhen Ning Tony He *State Key Laboratory of Genetics and Development of Complex Phenotypes, School of Life Sciences, MOE Engineering Research Center of Gene Technology, Shanghai Engineering Research Center of Industrial Microorganisms, Fudan University, Songhu Road 2005, Shanghai, 200438, China.ORCID http://orcid.org/0000-0003-4143-3219
Fangzhou Meng *State Key Laboratory of Genetics and Development of Complex Phenotypes, School of Life Sciences, MOE Engineering Research Center of Gene Technology, Shanghai Engineering Research Center of Industrial Microorganisms, Fudan University, Songhu Road 2005, Shanghai, 200438, China.
Xiaoyang QianState Key Laboratory of Genetics and Development of Complex Phenotypes, School of Life Sciences, MOE Engineering Research Center of Gene Technology, Shanghai Engineering Research Center of Industrial Microorganisms, Fudan University, Songhu Road 2005, Shanghai, 200438, China.
Yuying LiuState Key Laboratory of Genetics and Development of Complex Phenotypes, School of Life Sciences, MOE Engineering Research Center of Gene Technology, Shanghai Engineering Research Center of Industrial Microorganisms, Fudan University, Songhu Road 2005, Shanghai, 200438, China.
Xin FangState Key Laboratory of Genetics and Development of Complex Phenotypes, School of Life Sciences, MOE Engineering Research Center of Gene Technology, Shanghai Engineering Research Center of Industrial Microorganisms, Fudan University, Songhu Road 2005, Shanghai, 200438, China.
Dan YangDepartment of Orthopedics, Shanghai Children's Hospital, School of Medicine, Shanghai Jiao Tong University, Luding Road 355, Shanghai, 200062, China. yangdan@shchildren.com.cn.
Qing YangState Key Laboratory of Genetics and Development of Complex Phenotypes, School of Life Sciences, MOE Engineering Research Center of Gene Technology, Shanghai Engineering Research Center of Industrial Microorganisms, Fudan University, Songhu Road 2005, Shanghai, 200438, China. yangqing68@fudan.edu.cn.ORCID http://orcid.org/0000-0003-4162-922X

Funding

National Natural Science Foundation of China 82002839State Key Laboratory of Druggability Evaluation and Systematic Translational Medicine, Tianjin Medical University Cancer Institute & Hospital QZKF24-4Training Program for Emerging Clinical Scientists at Shanghai Children's Hospital 2024QK04
6 · The paper itself

Abstract

backgroundHydrogen sulfide (H

methodsApoptosis, cellular distribution of NR4A1, phosphorylation of IDO1, and the binding of phosphorylated IDO1 and SOCS3 are examined both in vitro and in vivo.

resultsIn this study, we confirm our hypothesis by showing that H

conclusionsH

Indexed as

ApoptosisHydrogen SulfideIndoleamine-Pyrrole 2,3,-DioxygenaseNuclear Receptor Subfamily 4, Group A, Member 1Proto-Oncogene Proteins c-bcl-2Signal TransductionSuppressor of Cytokine Signaling 3 ProteinAnimalsCell Line, TumorCell ProliferationGene Expression Regulation, NeoplasticHumansMiceMice, Inbred BALB CMice, NudePhosphorylationHydrogen SulfideIDO1 protein, humanIndoleamine-Pyrrole 2,3,-DioxygenaseNR4A1 protein, humanNuclear Receptor Subfamily 4, Group A, Member 1Proto-Oncogene Proteins c-bcl-2Suppressor of Cytokine Signaling 3 ProteinApoptosisHydrogen sulfideIndoleamine 2,3-dioxygenase 1Tumor therapy

Identifiers

PMID42129631
PMCPMC13374331

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.