Evidence map›Paper›PMID 42120807›Full record

ArticleBrain tumor pathology2026

Tectal glioma with pilocytic astrocytoma morphology carrying a histone H3 K27M mutation and malignant transformation: a literature review.

Moe Kumai, Masayuki Kanamori, Yoshiteru Shimoda, Kaishi Satomi, Yoshinari Osada, Shota Yamashita, Maako Kawamura, Asami Kodera, Hidetaka Niizuma, Yuto Yamazaki and 5 more

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Article in Brain tumor pathology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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0citing papers in PubMed
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1 · What the graph read from it

What it found

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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

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3 · Its place in the literature

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4 · The record

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5 · Who and what money

Authors and funding

15 authors.

Moe KumaiDepartment of Neurosurgery, Tohoku University Graduate School of Medicine, Sendai, Japan.
Masayuki KanamoriDepartment of Neurosurgery, Tohoku University Graduate School of Medicine, Sendai, Japan. masayuki.kanamori.e8@tohoku.ac.jp.ORCID http://orcid.org/0000-0002-3380-1192
Yoshiteru ShimodaDepartment of Neurosurgery, Tohoku University Graduate School of Medicine, Sendai, Japan.
Kaishi SatomiDepartment of Pathology, Kyorin University, School of Medicine, Tokyo, Japan.
Yoshinari OsadaDepartment of Neurosurgery, Tohoku University Graduate School of Medicine, Sendai, Japan.
Shota YamashitaDepartment of Neurosurgery, Tohoku University Graduate School of Medicine, Sendai, Japan.
Maako KawamuraDepartment of Personalized Medicine Center, Tohoku University Hospital, Sendai, Japan.
Asami KoderaDepartment of Pediatrics, Tohoku University Graduate School of Medicine, Sendai, Japan.
Hidetaka NiizumaDepartment of Pediatrics, Tohoku University Graduate School of Medicine, Sendai, Japan.
Yuto YamazakiDepartment of Pathology, Tohoku University Hospital, Sendai, Japan.
Hirofumi WatanabeDepartment of Pathology, Tohoku University Hospital, Sendai, Japan.
Mika WatanabeDepartment of Pathology, Tohoku University Hospital, Sendai, Japan.
Koichi IchimuraDepartment of Pathology, Kyorin University, School of Medicine, Tokyo, Japan.
Junji ShibaharaDepartment of Pathology, Kyorin University, School of Medicine, Tokyo, Japan.
Hidenori EndoDepartment of Neurosurgery, Tohoku University Graduate School of Medicine, Sendai, Japan.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

We report a case with pilocytic astrocytoma (PA) morphology carrying a histone H3 K27M mutation (H3-K27M) that progressed to PA with anaplasia. A 34-year-old woman presented with headache due to obstructive hydrocephalus from a tectal lesion. She underwent subtotal tumor resection. Histology showed a biphasic architecture with piloid cells, consistent with PA. Five months later, she developed recurrence. A second, partial resection was performed, with pathology again indicating PA. Despite subsequent radiation and chemotherapy, the tumor progressed. A third resection was done 15 months after the second. This time, histology revealed PA with anaplasia. The patient died of progressive disease 25 months after initial treatment. KIAA1549::BRAF fusion, H3-K27M, and loss of p16 expression due to promoter methylation were detected in all specimens. In all specimens, the presence of NF1 Y2285fs*1 was confirmed by Sanger sequencing, with an increasing proportion of the aberrant sequence at the third resection. DNA methylation profiling revealed that the second and third specimens were classified as diffuse midline glioma, H3 K27-altered. This case suggests that the H3-K27M mutation, MAPK pathway activation, and loss of p16 expression contribute to tumorigenesis, while clonal proliferation of the tumor harboring the NF1 mutation may play a role in malignant progression.

Indexed as

AnaplasiaDiffuse midline gliomaH3 K27-alteredKIAA1549:BRAF fusionPilocytic astrocytomaTectum

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.