ReviewJournal of inflammation research2026
Elucidating the Role of Neutrophils in the Inflammatory Response to Behcet Syndrome.
Review in Journal of inflammation research, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
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Authors and funding
3 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Behçet syndrome (BS) is a chronic, relapsing, multisystem inflammatory disorder in which both innate and adaptive immune mechanisms contribute to disease pathogenesis. Among innate immune cells, neutrophils have emerged as key effectors driving tissue inflammation, endothelial injury, and immunothrombosis. Early studies demonstrated enhanced chemotaxis, oxidative burst, and a primed activation state of circulating neutrophils, whereas more recent evidence highlights excessive neutrophil extracellular trap (NET) formation as an important contributor to vascular inflammation and thrombosis. In addition, recent omics-based studies have provided novel insights into neutrophil heterogeneity, activation pathways, and disease-specific signatures in BS. This review summarizes current knowledge on the role of neutrophils and NETs in the pathogenesis of BS, summarizes available evidence on mechanisms of neutrophil activation, and highlights potential directions for future research.
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Registered trials
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