ArticleInfection and drug resistance2026
Molecular Epidemiology of Carbapenem-Resistant
Article in Infection and drug resistance, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
1 citing paper in PubMed.
- Clinical and Microbiological Profile ofInfection and drug resistance · 2026Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
8 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Purpose: Carbapenem-resistant Patients and Methods: Twelve non-duplicate clinical CRSM isolates were collected from a tertiary hospital between 2010 and 2024. Clinical data were retrospectively obtained from electronic medical records. Antimicrobial susceptibility testing was performed using the Vitek-2 system. Whole-genome sequencing (WGS) was conducted to determine resistance and virulence gene profiles, sequence types, and phylogenetic relationships. Pulsed-field gel electrophoresis and conjugation assays were performed to assess clonal relatedness and plasmid transferability. Results: CRSM infections predominantly occurred in neonates (50.0%) and adults (50.0%), with the respiratory tract as the most common infection site (66.7%). All isolates exhibited multidrug-resistant phenotypes; resistance to β-lactams ranged from 50.0% to 100.0%, while no amikacin-resistant isolates were detected. Two dominant clonal lineages were identified: ST595 (5 strains) uniformly carrying Conclusion: CRSM dissemination in this hospital is likely driven by two co-circulating high-risk clones (ST595-
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.