Evidence map›Paper›PMID 42102059›Full record

ArticlePloS one2026

A pumpless liver-adipose model for studying metabolic dysfunction and drug responses.

Zeinab Ebrahimian, Fatemeh Kalalinia, Amir Reza Ameri, Hossein Hosseinzadeh, Bibi Marjan Razavi, Seyed Ali Mousavi Shaegh

Abstract read
In one paragraph

Article in PloS one, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Zeinab EbrahimianDepartment of Pharmacodynamics and Toxicology, School of Pharmacy, Mashhad University of Medical Sciences, Mashhad, Iran.
Fatemeh KalaliniaBiotechnology Research Center, Pharmaceutical Technology Institute, Mashhad University of Medical Sciences, Mashhad, Iran.
Amir Reza AmeriLaboratory for Microfluidics and Medical Microsystems, Research Institute for Medical Sciences, Mashhad University of Medical Sciences, Mashhad, Iran.
Hossein HosseinzadehDepartment of Pharmacodynamics and Toxicology, School of Pharmacy, Mashhad University of Medical Sciences, Mashhad, Iran.
Bibi Marjan RazaviDepartment of Pharmacodynamics and Toxicology, School of Pharmacy, Mashhad University of Medical Sciences, Mashhad, Iran.
Seyed Ali Mousavi ShaeghLaboratory for Microfluidics and Medical Microsystems, Research Institute for Medical Sciences, Mashhad University of Medical Sciences, Mashhad, Iran.ORCID https://orcid.org/0000-0001-8770-3267

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Metabolic dysfunction is a primary driver of chronic diseases, such as obesity and type 2 diabetes. Developing in vitro models allows to understand metabolic mechanisms and develop effective treatments. To this end, a pumpless open-top co-culture (POCC) device to simulate liver-adipose tissue interactions was developed. The POCC comprises a four-chamber bioreactor with an open-top configuration and an easy-to-assemble cover cap. Rotational motion enabled perfusion of culture media among the interconnected chambers. HepG2 and 3T3-L1 cells were co-cultured and exposed to olanzapine (Olz), chlorogenic acid (CGA), and metformin (Met) for four days. Drug responses in the POCC were compared with those in 96-well monocultures. Assessments were conducted for glucose/triglyceride (TG) levels, lipid accumulation, and cell viability. While viability remained unchanged, Olz increased lipid content, extracellular glucose and TG levels in the POCC model, effects that CGA and Met mitigated. This practical and physiologically relevant platform offers a promising alternative for preclinical screenings that could be employed to study other multi-tissue interactions in various disease models.

Indexed as

Adipose TissueLiver3T3-L1 CellsAnimalsBioreactorsCell SurvivalCoculture TechniquesGlucoseHep G2 CellsHumansLipid MetabolismMetforminMiceModels, BiologicalOlanzapineTriglyceridesGlucoseMetforminOlanzapineTriglycerides

Identifiers

PMID42102059
PMCPMC13155575

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.