Evidence map›Paper›PMID 42100250›Full record

ReviewFrontiers in medicine2026

BACH transcription factors in diseases of the exocrine and endocrine pancreas: effects, mechanisms, and clinical implications.

Robert Jaster

Abstract readReview
In one paragraph

Review in Frontiers in medicine, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author.

Robert JasterDivision of Gastroenterology, Hepatology and Nutritional Medicine, Department of Internal Medicine, Rostock University Medical Center, Rostock, Germany.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The BTB and CNC homology (BACH) proteins BACH1 and BACH2 are highly conserved transcriptional repressors that exert their effects by forming heterodimers with small musculoaponeurotic fibrosarcoma (MAF) proteins. BACH1 is ubiquitously expressed and functions by competing with nuclear factor erythroid 2-like 2 (NRF2) for binding to MAF. By suppressing the transcription of heme oxygenase 1 (

Indexed as

BACH1BACH2diabetes mellituspancreatic cancerpancreatitis

Identifiers

PMID42100250
PMCPMC13143592

What OpenQuestion holds

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LicenceCC BY
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.