ArticleFrontiers in immunology2026
Susceptibility of mice to primary
Article in Frontiers in immunology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
1 citing paper in PubMed.
- Proteomic reprogramming of ileal epithelial cells during homologous superimposed intestinal trematode infection reveals coordinated restoration of intestinal homeostasis.PLoS neglected tropical diseases · 2026Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
5 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Background: Helminth infections are among the most prevalent neglected tropical diseases, causing an enormous impact in health and the socioeconomic status of developing countries. The study host-parasite interactions constitute a promising approach for developing new tools to control these infections. Methods: Male ICR mice were infected with Results: The infection induced strong upregulation of proteins involved in mitochondrial oxidative phosphorylation, purine metabolism, and exopeptidases activating the renin-angiotensin system, fostering oxidative stress and inflammation. Downregulated proteins included those linked to fatty acid β-oxidation, nucleotide binding, vesicular trafficking, and tight junction maintenance, indicating impaired epithelial homeostasis. Antimicrobial peptides were overexpressed, while eosinophil- and neutrophil-derived effectors were reduced, suggesting a skewed Th1-biased profile. Conclusion: The primary
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Registered trials
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