ReviewFrontiers in cell and developmental biology2026
Targeted autophagy: research progress on the regulatory potential of periodontitis prevention and treatment.
Review in Frontiers in cell and developmental biology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Abstract
Chronic periodontitis is a multifactorial inflammatory disease primarily caused by bacterial pathogens, leading to progressive destruction of both soft and hard periodontal tissues. While dental plaque biofilm has been universally recognized as the primary etiological factor in periodontitis development, disease progression is also influenced by local irritants and systemic modulators, making its pathogenesis complex and widely studied. Autophagy, an essential cellular self-protection mechanism, plays a pivotal role in maintaining intracellular homeostasis, responding to stress stimuli, and defending against microbial invasion. Recently, this process has emerged as a key research focus in periodontal pathology. This study investigates the dynamic changes and functional variations of autophagy during different stages of periodontitis, aiming to elucidate its precise role in disease initiation and progression.
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