ArticleClinical, cosmetic and investigational dermatology2026
Genetically Predicted t
Article in Clinical, cosmetic and investigational dermatology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
4 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Purpose: Bullous pemphigoid (BP) is an autoimmune blistering disease linked to T-lymphocyte dysregulation. Adenosine deaminase (ADA) and adenosine metabolites modulate T-cell function, suggesting a potential role in BP pathogenesis. However, causal evidence from human genetic studies is lacking. This study aimed to investigate the potential causal associations of genetically predicted levels of ADA, ADA protein, and several adenosine metabolites with the risk of BP and its subtypes (mucous membrane pemphigoid [MMP] and other/unspecified pemphigoid [OUP]). Patients and Methods: We conducted a two-sample Mendelian randomization (MR) study using summary statistics from large-scale genome-wide association studies (GWAS). Genetic instruments for ADA levels, ADA protein levels, 5-methylthioadenosine, N1-methyladenosine, N6-carbamoylthreonyladenosine (t Results: Genetically predicted higher t Conclusion: This MR study suggests a potential causal association between higher t
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.