ArticleAntioxidants (Basel, Switzerland)2026
The c-Abl-RIPK3 Axis Drives Mitochondrial Dysfunction and Impaired Mitophagy in Gaucher Disease Models.
Cristian M Lamaizon, Renatta Tironi-Hernández, Nohela B Arévalo, Sebastián D Ahumada, Daniela A Gutiérrez, Laura Brito-Fernández, Andrea Del Campo, Silvana Zanlungo, Alejandra R Álvarez
Abstract read
In one paragraphArticle in Antioxidants (Basel, Switzerland), 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from itWhat it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registryThe trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
3 · Its place in the literatureWho cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
4 · The recordCorrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what moneyAuthors and funding
9 authors.
Cristian M LamaizonCell Signaling Laboratory, Biological Sciences Faculty, Pontificia Universidad Católica de Chile, Santiago 8331150, Chile.ORCID 0000-0001-8515-3804 Renatta Tironi-HernándezCell Signaling Laboratory, Biological Sciences Faculty, Pontificia Universidad Católica de Chile, Santiago 8331150, Chile.
Nohela B ArévaloCell Signaling Laboratory, Biological Sciences Faculty, Pontificia Universidad Católica de Chile, Santiago 8331150, Chile.
Sebastián D AhumadaCell Signaling Laboratory, Biological Sciences Faculty, Pontificia Universidad Católica de Chile, Santiago 8331150, Chile.
Daniela A GutiérrezCell Signaling Laboratory, Biological Sciences Faculty, Pontificia Universidad Católica de Chile, Santiago 8331150, Chile.ORCID 0000-0001-5823-8044 Laura Brito-FernándezDepartment of Gastroenterology, Medicine Faculty, Pontificia Universidad Católica de Chile, Santiago 8320165, Chile.
Andrea Del CampoPhysiology and Cell Bioenergetics Laboratory, Pharmacy School, Chemistry and Pharmacy Faculty, Pontificia Universidad Católica de Chile, Santiago 7820436, Chile.
Silvana ZanlungoDepartment of Gastroenterology, Medicine Faculty, Pontificia Universidad Católica de Chile, Santiago 8320165, Chile.ORCID 0000-0001-8383-9829 Alejandra R ÁlvarezCell Signaling Laboratory, Biological Sciences Faculty, Pontificia Universidad Católica de Chile, Santiago 8331150, Chile.
Funding
Agencia Nacional de Investigación y Desarrollo 202045Fondo Nacional de Desarrollo Científico y Tecnológico 1230337Fondo Nacional de Desarrollo Científico y Tecnológico 1230428Fondo Nacional de Desarrollo Científico y Tecnológico 1241503Fundación Nacional para el Desarrollo 25I10566
6 · The paper itselfAbstract
Gaucher disease (GD) is characterized by the accumulation of glucosylceramide within lysosomes due to mutations in the
Indexed as
autophagyc-AblGaucher diseaselysosomal storage disordersmitochondrial dysfunctionmitophagyoxidative stressRIPK3
Identifiers
PMID42072107
PMCPMC13113124
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