ReviewCurrent psychiatry reports2026
Structural Neuroimaging in Substance-Induced Psychosis.
Review in Current psychiatry reports, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
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Authors and funding
2 authors.
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Abstract
purpose of reviewThis review evaluates structural neuroimaging research on substance-related psychosis to determine if these conditions share common neurobiological pathways with primary psychotic disorders, such as schizophrenia, or exhibit unique signatures. RECENT
findingsResearch indicates that methamphetamine, cannabis, and cocaine induce significant structural changes, including cortical thinning and grey matter loss. Methamphetamine-induced psychosis is associated with frontal lobe reductions and smaller hippocampal and amygdalar volumes. Cannabis use is linked to alterations in the cingulate cortex and cerebellar networks, particularly when use begins early in life. While many neuroimaging patterns overlap with schizophrenia substance-specific variations exist. For instance, methamphetamine users show more pronounced amygdalar reduction than those with primary psychosis. Generally, structural deficits are more severe and widespread in schizophrenia than in purely substance-induced cases. Substance-induced psychoses exhibit neuroanatomical signatures largely similar to primary psychoses, though distinct regional variations suggest partially divergent mechanisms. Future longitudinal and multimodal research is essential to identify definitive biomarkers for clinical differentiation.
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