ArticleAnatomy & cell biology2026
Distinct delayed development of the basisphenoid-ethmoid interface in human fetuses: a histological study using human fetuses.
Article in Anatomy & cell biology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
In non-human primates, the presphenoid-ethmoid synchondrosis (PSept) functions as a major growth center of the midline skull base, yet whether humans possess a developmental analogue remains unresolved. To clarify whether a human counterpart exists, we re-examined the basisphenoid-ethmoid (SPH-ETH) junction using sagittal histological sections from 20 human fetuses (11-39 weeks) preserved in the Akita University collection. At 15-16 weeks, chondrocytes in the anterior portion of the SPH-immediately anterior to the pituitary fossa-became slender or irregularly shaped with increased extracellular matrix, forming a distinct low-density zone (<8 cells per 0.1 mm
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