ArticleACS chemical biology2026
Selective Targeting of Protein Arginine Methyltransferase 1 (PRMT1) Mutant Activity by a Small Molecule Activation Strategy.
Article in ACS chemical biology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
Protein arginine methyltransferase 1 (PRMT1) is involved in the post-translational modification of cellular proteins in higher organisms by adding a methyl group to arginine-containing substrates. Various studies have demonstrated that the overexpression of PRMT1 is common in cancer development and correlates with poor survival prognosis of patients. Selective targeting of individual PRMT isoforms with chemical inhibitors is technically challenging due to the high sequence similarity among them. This study aims to create a distinct method to selectively modulate PRMT1 activity with small molecules. In our approach, enzymatically inactive PRMT1 forms were produced by mutating residue H293 to alanine or glycine by site-directed mutagenesis. Next, an exogenous chemical molecule was used to activate the enzymatic activity of the silent PRMT1 form by introducing a structurally complementary functional group at the mutation site. We demonstrated that the small molecule compound 4-methylimidazole effectively enhanced PRMT1-H293G activity by 8.6-fold while not affecting the enzymatic activities of the other PRMT isoforms. The selective enhancement effect of small molecule activation of PRMT1 was observed on both single-substrate and complex proteome mixtures. Hence, we presented a unique chemical genetics strategy to modulate PRMT1 activity with small molecules with high isoform selectivity, which can be of great potential to be applied to dissect cellular functions of individual particular PRMT members with fast onset and in a real-time manner.
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