ArticleClinical cardiology2026
Admission Shock Index Is an Independent Predictor of In-Hospital All-Cause Mortality in Patients With Acute Aortic Dissection and Intramural Hematoma.
Article in Clinical cardiology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
2 citing papers in PubMed.
- Admission Shock Index in Acute Aortic Dissection: A Triage Trigger or a Restated Vital Sign?Clinical cardiology · 2026Article
- Admission Shock Index Is an Independent Predictor of In-Hospital All-Cause Mortality in Patients With Acute Aortic Dissection and Intramural Hematoma.Clinical cardiology · 2026Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
9 authors.
Funding
Abstract
backgroundAcute aortic dissection (AD) and intramural hematoma (IMH) are associated with high mortality, necessitating reliable early risk prediction. The shock index (SI) is a potential prognostic marker in critical care, but its value in AD/IMH remains unclear. This study evaluated the association between admission SI and in-hospital all-cause mortality.
methodsThis single-center retrospective cohort study included 1250 patients with acute AD/IMH, stratified by an optimal SI cut-off of 0.6 determined by ROC analysis. Kaplan-Meier curves and Cox proportional hazards models were used to assess the relationship. Subgroup analyses were also conducted to confirm the consistency of the main findings.
resultsThe 30-day cumulative in-hospital all-cause mortality was significantly higher in the SI ≥ 0.6 group than in the SI < 0.6 group (Total: 25.7% vs. 14.4%, p < 0.001; Stanford A: 35.5% vs. 25.2%, p < 0.001; Stanford B: 13.4% vs. 4.8%, p < 0.001). An SI ≥ 0.6 was independently associated with increased in-hospital mortality (adjusted hazard ratio (aHR) 1.67, p = 0.004), consistent across Stanford A (aHR 1.52, p = 0.038) and Stanford B (aHR 2.57, p = 0.014) subgroups. Furthermore, the association was stronger among patients managed without surgery or thoracic endovascular aortic repair (TEVAR) (Total: aHR 2.02, p < 0.001; Stanford A: aHR 1.77, p = 0.009; Stanford B: aHR 3.30, p = 0.004).
conclusionAn admission SI ≥ 0.6 is independently associated with increased in-hospital all-cause mortality in acute AD/IMH, particularly among those managed without surgery/TEVAR. Admission SI may serve as a simple, rapid, and valuable tool for early clinical risk stratification.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.