ArticleFrontiers in medicine2026
Article in Frontiers in medicine, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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6 authors.
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Abstract
Background: The 17q22 deletion syndrome is associated with a range of clinical phenotypes, primarily due to differences in the size and position of the deleted genomic segment. In all previously reported cases, diagnoses were made postnatally, with clinical manifestations such as intellectual disability, visual impairment, and other neurological abnormalities. Methods: Karyotype analysis, chromosomal microarray analysis, and whole-exome sequencing were performed to identify the genetic etiology of a fetus with nuchal pellucida thickening. Summarize the previously reported cases of similar 17q22 deletions and conduct a systematic review. Results: Chromosomal microarray analysis revealed an arr[GRCh37] 17q22(55,501,204_56,476,808) × 1, which was confirmed as a Conclusion: Integrating the genetic findings in this fetus, the abnormal ultrasound structural phenotype, and the clinical characteristics of existing cases, we conclude that
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