Evidence map›Paper›PMID 42031225›Full record

ReviewInternational journal of radiation oncology, biology, physics2026

Mechanisms, Microenvironments, and Models: Understanding Therapeutic Resistance in Glioblastoma.

Amy J Wisdom, Heidi Temple, Yufei Cui, Cassandra Tafuri Del Vecchio, Tiffany Kim, Forest M White, Scott R Floyd, Stefani Spranger

Abstract readReview
In one paragraph

Review in International journal of radiation oncology, biology, physics, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed.

  1. Review
  2. Review
  3. Article
  4. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Amy J WisdomDepartment of Radiation Oncology, Massachusetts General Hospital, Boston, Massachusetts; Department of Biology, Massachusetts Institute of Technology, Cambridge, Massachusetts; David H. Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, Massachusetts. Electronic address: amy_wisdom@dfci.harvard.edu.
Heidi TempleDepartment of Biology, Massachusetts Institute of Technology, Cambridge, Massachusetts; David H. Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, Massachusetts.
Yufei CuiDavid H. Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, Massachusetts; Department of Biological Engineering, Massachusetts Institute of Technology, Cambridge, Massachusetts.
Cassandra Tafuri Del VecchioDepartment of Biology, Massachusetts Institute of Technology, Cambridge, Massachusetts.
Tiffany KimDepartment of Biology, Massachusetts Institute of Technology, Cambridge, Massachusetts.
Forest M WhiteDavid H. Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, Massachusetts; Department of Biological Engineering, Massachusetts Institute of Technology, Cambridge, Massachusetts.
Scott R FloydDepartment of Radiation Oncology, Duke University, Durham, North Carolina.
Stefani SprangerDepartment of Biology, Massachusetts Institute of Technology, Cambridge, Massachusetts; David H. Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, Massachusetts.

Funding

Quantitative systems biology of glioblastoma cells and their interactions with the neuronal and immunological milieuU54CA283114 · NCI · MASSACHUSETTS INSTITUTE OF TECHNOLOGY · PI Forest M White · 2023 to 2026
$9.7M
NCI NIH HHS U54 CA283114
6 · The paper itself

Abstract

Glioblastoma (GBM) is the most common and lethal primary malignant brain tumor in adults. Despite aggressive multimodal therapy, including maximal safe resection, radiation therapy, and temozolomide chemotherapy, median survival remains approximately 16 months, and nearly all tumors recur. Over the past 2 decades, numerous therapies that demonstrated promise in preclinical studies have failed to improve outcomes in randomized clinical trials, underscoring the therapeutic resistance that defines this disease. This resistance arises from the convergence of tumor-intrinsic mechanisms, microenvironmental constraints, and limitations of current preclinical models. In this review, we synthesize advances in understanding the molecular, cellular, and anatomic determinants of resistance to radiation therapy, chemotherapy, targeted therapies, and immunotherapies in adult GBM. We highlight how extensive intra- and intertumoral heterogeneity, transcriptional plasticity, and adaptive reprogramming enable tumor cells to evade cytotoxic stress. Key resistance mechanisms include activation of DNA damage response pathways, exploitation of hypoxic niches, therapy-induced mesenchymal transitions, and evasion of immune surveillance through impaired antigen presentation and a profoundly immunosuppressive tumor microenvironment. We further discuss how GBM exploits the unique immunologic features of the central nervous system, including the blood-brain barrier, limited antigen burden, and tolerogenic myeloid populations, to blunt the efficacy of immunotherapies. A major focus of this review is the role of preclinical models in shaping our understanding of therapeutic resistance. We critically evaluate established cell lines, patient-derived xenografts, syngeneic models, and genetically engineered mouse models, emphasizing both their strengths and their inability to fully recapitulate defining features of human GBM. Finally, we outline emerging strategies to overcome resistance, including rational combination therapies, adaptive trial designs, improved biomarker-driven stratification, and integrative modeling approaches. Together, these insights provide a framework for translating mechanistic understanding into more effective, durable therapies for glioblastoma.

Indexed as

Brain NeoplasmsDrug Resistance, NeoplasmGlioblastomaRadiation ToleranceTumor MicroenvironmentAnimalsDNA DamageHumansImmunotherapyTumor Escape

Identifiers

PMID42031225
PMCPMC13224335

What OpenQuestion holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.