Evidence map›Paper›PMID 42029708›Full record

ReviewDiabetologia2026

Perspectives on human adipose tissue: from cellular mechanisms to clinical complications.

Mikael Rydén

Abstract readReview
In one paragraph

Review in Diabetologia, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author.

Mikael RydénDepartment of Medicine Huddinge, Karolinska Institutet, Karolinska University Hospital, Stockholm, Sweden. mikael.ryden@ki.se.

Funding

ERC SynergyKnut och Alice Wallenbergs Stiftelse Clinical Scholar
6 · The paper itself

Abstract

White adipose tissue (WAT) is a remarkably plastic organ integrating metabolic, endocrine and immune functions. In humans, perturbations in adipocyte turnover, size regulation and cellular heterogeneity underpin the transition from metabolic health to obesity-related disease. This review, based on the 2025 Camillo Golgi Prize Lecture, highlights selected mechanistic and translational insights, primarily from our work and that of close collaborators, that have contributed to reshaping our understanding of human adipose biology.

Indexed as

Adipose TissueAdipose Tissue, WhiteObesityAdipocytesAdipogenesisAnimalsDiabetes Mellitus, Type 2HumansInsulin ResistanceLipid MetabolismAdipocytesAdipogenesisDepotsDiabetesInflammationInsulin resistanceObesityReviewSingle-cell transcriptomics

Identifiers

PMID42029708
PMCPMC13236842

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.