ArticleHealth science reports2026
Association Between Autoimmune Thyroiditis and Cervical Artery Dissection: A Retrospective Cohort Study.
Article in Health science reports, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
1 citing paper in PubMed.
- Association Between Autoimmune Thyroiditis and Cervical Artery Dissection: A Retrospective Cohort Study.Health science reports · 2026Article
Corrections and comments
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Authors and funding
3 authors.
Funding
Abstract
Background and Aims: Limited evidence suggests autoimmune thyroiditis (AT) could represent a risk factor for cervical artery dissection (CeAD). We tested the hypothesis of a positive association between AT and CeAD within 3 years following diagnosis compared to matched euthyroid controls. Methods: We searched a de-identified United States' data resource (TriNetX LLC) for adults age ≥ 18 with newly-diagnosed AT, and euthyroid controls, from 2014 to 2024. Propensity matching controlled for variables associated with CeAD. Our primary outcome was the risk ratio (RR) for CeAD within 3 years' follow-up. Secondary outcomes explored the cumulative incidence of CeAD and RRs for vertebral and carotid artery dissection and stroke. Results: After matching, there were 143,831 patients per cohort (80% female). Comparing the AT cohort to matched euthyroid controls, the incidence and risk of CeAD was significantly greater [95% CI] (0.040% vs. 0.025%; RR = 1.58 [1.04, 2.40]; Conclusion: The present findings suggest that AT is a risk factor for CeAD. Additional research is needed to corroborate these findings and clarify factors that may mediate the AT-CeAD association, such as medications, thyroid hormones, and antibody levels.
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Registered trials
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