ArticleCureus2026
When Feeding Tubes Cause Biliary Disease: A Case of Acalculous Cholecystitis Due to Gastrostomy Tube Obstruction.
Article in Cureus, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
3 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Acute acalculous cholecystitis (AAC) is an inflammatory condition of the gallbladder that occurs in the absence of gallstones and is increasingly recognized among medically complex and long-term care populations. Patients receiving chronic enteral nutrition via gastrostomy tubes may be at risk for biliary complications related to impaired gallbladder emptying or mechanical factors. We describe an 81-year-old female nursing home resident with a previous medical history of hypertension, diabetes mellitus, Alzheimer's dementia, hypothyroidism, and gastrostomy tube dependence who presented with five days of malaise, nausea, vomiting, and right upper quadrant pain. Laboratory evaluation demonstrated a cholestatic liver enzyme pattern without leukocytosis. Bedside ultrasonography revealed gallbladder dilation without gallstones. Contrast-enhanced computed tomography of the abdomen and pelvis demonstrated migration of the balloon-retained gastrostomy tube into the proximal duodenum, causing extrinsic compression of the distal common bile duct, with associated gallbladder distention and intra- and extrahepatic biliary dilatation. Magnetic resonance cholangiopancreatography confirmed obstructive physiology without choledocholithiasis. The gastrostomy tube was deflated, removed, and repositioned, resulting in rapid clinical and biochemical improvement with conservative management. In patients receiving long-term enteral nutrition, reduced physiologic gallbladder stimulation may predispose to bile stasis, creating vulnerability to obstruction when tube malposition occurs. Imaging plays a central role in diagnosis, and early recognition with prompt tube repositioning can reverse obstruction and prevent progression to more severe gallbladder injury. Clinicians should be aware that, in patients with gastrostomy tube dependence, unexplained cholestatic liver enzyme elevation should prompt assessment of tube position and evaluation for possible device-related biliary obstruction.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.