ArticleKidney international reports2026
The Diversity of Transplant Glomerulitis and its Relationship to Alloantibody.
Article in Kidney international reports, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
1 citing paper in PubMed.
- Transplant Glomerulitis: Paying Due Deference to T-cell Mediated Rejection and Antibody Independent Mechanisms.Kidney international reports · 2026Article
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Authors and funding
4 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Introduction: Although transplant glomerulitis often signifies antibody-mediated rejection (AMR), the pathophysiology of complement degradation split-product 4d (C4d) staining of peritubular capillaries (C4d Methods: We compared Banff 1997 defined glomerulitis ( Results: Epidemiological risk factors were female recipients, living donation, early T-cell-mediated rejection (TCMR) or AMR, and previous corticosteroid rejection treatment. Glomerulitis associated with sensitization in 38.0%, ischemic endothelial injury in 20.7%, iatrogenic minimization in 30.3%, and nonadherence in 21.4%. Banff g score correlated with DSA+, C4d Conclusion: Glomerulitis is a heterogenous biological lesion that frequently signifies AMR, however TCMR or innate cellular inflammation caused many DSA- and/or C4d
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