ArticleFrontiers in plant science2026
Natural variation of the
Article in Frontiers in plant science, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
11 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
100-seed weight (100-SW) is a critical determinant of soybean yield. The identification and functional characterization of its underlying genes are therefore essential for the genetic improvement of seed and yield-related traits. A residual heterozygous line (RHL) segregating for 100-SW was derived from a recombinant inbred line (RIL) population generated by crossing small-seed (19.75 ± 1.93 g) and large-seed (26.20 ± 0.82 g) soybean parents. Phenotypic segregation of 100-SW was analyzed, and Chi-square test was used to verify the segregation ratio. Bulked segregant analysis combined with whole-genome sequencing (BSA-seq) was performed using both Euclidean distance and index algorithms to map the target gene. Functional annotation, molecular marker validation, and germplasm resequencing were conducted to identify the key candidate gene and its haplotypes. Phenotypic analysis showed significant segregation and normal distribution of 100-SW in the RHL, with a Chi-square-verified 1:2:1 segregation ratio, indicating control by a single nuclear gene. BSA-seq mapped the gene to a 5.46 Mb region on chromosome 19, where 74 non-synonymous SNPs in coding sequences were identified (including one causing initiation codon loss), distributed across 36 genes.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.