ArticleFEBS open bio2026
Anchorage-independent and faster growth in clonal population from UV-irradiated NER-deficient cells.
Article in FEBS open bio, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Authors and funding
7 authors.
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Abstract
Human tumours are characterized by both molecular and functional heterogeneity, which drives cancer cell growth. In this context, the selection of subclones that acquire enhanced proliferative capacity and resistance to pharmacological treatments represents a necessary step in tumour progression. In the presence of DNA damage, efficient signalling and lesion removal are crucial in preventing the accumulation of genomic mutations. In Nucleotide Excision Repair (NER) pathways, the loss of interaction between DDB2 and PCNA (referred to as DDB2
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