ArticleAnnals of intensive care2026
Pulmonary pulsatility quantified by electrical impedance tomography in severe acute respiratory distress syndrome patients undergoing extracorporeal membrane oxygenation support.
Article in Annals of intensive care, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
Background: The cardiac-related pulsatility signal from electrical impedance tomography (EIT) correlates with stroke volume in mechanically ventilated patients with acute respiratory distress syndrome (ARDS). However, in swine models, regional pulsatility amplitude was also shown to increase with downstream flow obstruction. We aimed to investigate the relationship between regional pulsatility and pulmonary hemodynamics in a cohort of severe ARDS patients on veno-venous extracorporeal membrane oxygenation (ECMO). Methods: We reanalysed data obtained from 20 ARDS patients receiving ECMO support. EIT was recorded 30 min after adjusting ECMO blood flow to target three ranges of mixed venous oxygen saturation (SvO Results: Across blood flow steps, pulsatility amplitude was directly related to stroke volume (SV) (β = 0.28 (0.06 - 0.5) ml*/mL, p = 0.014) and systolic pulmonary artery pressure (PAPs) (β = 0.47 (0.14 - 0.81) ml*/mmHg, p = 0.008) and inversely related to mixed venous oxygen tension (PvO Conclusion: n severe ARDS patients on ECMO, pulsatility amplitude reflects stroke volume changes induced by positive intrathoracic pressures and mixed venous saturation targets. However, downstream flow obstruction appears to be the leading determinant in the dorsal lung and may be useful to monitor right heart loading in patients with ARDS.
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