Article in Nature communications, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registry
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what money
Authors and funding
19 authors.
Kerstin FilippiInstitute of Physiology I, Medical Faculty, University of Bonn, Bonn, Germany.
Kathrin Graf-RiesenInstitute of Physiology I, Medical Faculty, University of Bonn, Bonn, Germany.
Maithreyan KuppusamyCentral Institute for Engineering, Electronics and Analytics, ZEA-3, Forschungszentrum Jülich, Jülich, Germany.ORCID 0000-0001-6866-0417
Andreas UngerInstitute of Physiology II, University of Muenster, Muenster, Germany.
Kenichi KimuraLife Science Center for Survival Dynamics, Tsukuba Advanced Research Alliance (TARA), University of Tsukuba, Ibaraki, 305-8577, Japan.ORCID 0000-0002-0363-8865
Martin MatijassCentral Institute for Engineering, Electronics and Analytics, ZEA-3, Forschungszentrum Jülich, Jülich, Germany.
Henrique BaetaCentral Institute for Engineering, Electronics and Analytics, ZEA-3, Forschungszentrum Jülich, Jülich, Germany.
Magdalena PodlachaDepartment of Molecular Biology, Faculty of Biology, University of Gdańsk, Gdańsk, Poland.ORCID 0000-0002-7709-5955
Daniel HaertterInstitute of Pharmacology and Toxicology, University Medical Center Göttingen, Göttingen, Germany.ORCID 0000-0002-9582-6141
Alexei P KudinInstitute of Experimental Epileptology and Neurocognition, University Bonn Medical Center, 53105, Bonn, Germany.
Martin WiemannInstitute of Physiology I, Medical Faculty, University of Bonn, Bonn, Germany.
Grzegorz WęgrzynDepartment of Molecular Biology, Faculty of Biology, University of Gdańsk, Gdańsk, Poland.ORCID 0000-0003-4042-7466
Cornelia KornblumDepartment of Neurology, University Bonn Medical Center, 53105, Bonn, Germany.ORCID 0000-0002-0111-7281
Jens ReimannDepartment of Neurology, University Bonn Medical Center, 53105, Bonn, Germany.
Wolfgang A LinkeInstitute of Physiology II, University of Muenster, Muenster, Germany.ORCID 0000-0003-0801-3773
Pitter F HuesgenCentral Institute for Engineering, Electronics and Analytics, ZEA-3, Forschungszentrum Jülich, Jülich, Germany.ORCID 0000-0002-0335-2242
Wolfram S KunzInstitute of Experimental Epileptology and Neurocognition, University Bonn Medical Center, 53105, Bonn, Germany.ORCID 0000-0003-1113-3493
Bernd K FleischmannInstitute of Physiology I, Medical Faculty, University of Bonn, Bonn, Germany.ORCID 0000-0002-9202-8363
Michael HesseInstitute of Physiology I, Medical Faculty, University of Bonn, Bonn, Germany. mhesse1@uni-bonn.de.ORCID 0000-0002-7518-0224
Funding
Deutsche Forschungsgemeinschaft (German Research Foundation) 388932620
6 · The paper itself
Abstract
Myofibrillar myopathy 6 is a rare, autosomal-dominant neuromuscular disorder caused by an amino acid exchange Pro209Leu in the co-chaperone BAG3, which disrupts muscle protein turnover and causes severe muscle weakness and shortened lifespan. We generated transgenic mice overexpressing the human mutant BAG3
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.
Blockage of autophagy causes severe skeletal muscle disruption in a mouse model for myofibrillar myopathy 6. · full record | OpenQuestion