ArticleEuropean journal of population = Revue europeenne de demographie2026
A Double Decomposition of Standard Deviation Below the Modal Age at Death and the Role of Causes of Death.
Article in European journal of population = Revue europeenne de demographie, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
1 citing paper in PubMed.
- Mortality Dynamics in Low-Mortality Countries: An Introduction to the Special Issue.European journal of population = Revue europeenne de demographie · 2026Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
3 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Lifespan inequality is a fundamental indicator of population health, reflecting inequalities in the timing of death. Life expectancy-based indicators have been widely used to monitor changes in lifespan variation across populations. This study proposes using indicators relative to the modal age at death (M): the standard deviation below the mode, [Formula: see text], which captures variation in premature mortality, and the standard deviation above the mode, [Formula: see text], which reflects variation in senescent mortality. Although trends in [Formula: see text] are relatively well documented, less is known about how [Formula: see text] has changed over time and what drives these changes. This study aims to (1) document and compare trends in [Formula: see text] and [Formula: see text] across high-income countries since 1960, and (2) examine the contribution of cause-specific mortality to changes in [Formula: see text] in selected countries. To achieve this, we propose a novel two-step decomposition method. In the first step, changes in [Formula: see text] are decomposed into two components: one attributable to shifts in the modal age itself ("mode" component) and another to changes in the shape of the age-at-death distribution ("distribution" component). In the second step, the "distribution" component is further decomposed by cause of death. Applying this framework to data from Japan and the U.S., results revealed that the decline in [Formula: see text] in the U.S. was primarily driven by reductions in heart disease and neoplasm mortality. However, these gains were partially offset by increased variation linked to infectious diseases and external causes. In Japan, declines in [Formula: see text] were primarily driven by reductions in cerebrovascular diseases, heart disease (women), and neoplasms (men), while increases in variation since the mid-1990s were largely attributable to external causes and neoplasms (women). This decomposition is a useful tool for identifying the factors that drive or hinder the compression of premature mortality.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.