ArticleActa neuropathologica communications2026
Meningeal and infiltrating T-cells are associated with repetitive head trauma and tau-mediated neurodegeneration in chronic traumatic encephalopathy.
Article in Acta neuropathologica communications, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
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1 citing paper in PubMed.
- Mild traumatic brain injury promotes chronic cerebrovascular inflammation and glymphatic suppression.Molecular neurodegeneration advances · 2026Article
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10 authors.
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Abstract
Central nervous system (CNS) associated T-cells are present in the meninges and perivascular spaces of healthy brain tissue, but their recruitment into the brain parenchyma is increased by inflammation and hyperphosphorylated tau (p-tau) accumulation. Chronic traumatic encephalopathy (CTE) is a progressive tauopathy associated with exposure to repetitive head impacts (RHI) and definitively diagnosed by the presence of a pathognomonic perivascular p-tau lesion, most commonly at the sulcal depths of the dorsolateral frontal cortex (DLF). Exposure to RHI and CTE is associated with substantial neuroinflammation; however, the involvement of T cells is unknown. Here, we used post-mortem human brain tissue to assess T-cell accumulation in the DLF of 58 individuals exposed to RHI, including 19 with neuropathologically verified Low CTE (stage I-II), 23 with neuropathologically verified High CTE (stage III-IV), and 16 without CTE, as well as 18 controls unexposed to RHI and without CTE. Multiplex immunofluorescence was utilized to label T-cells, microglia, p-tau, and synapses in the leptomeninges, sulcal gray matter, crest gray matter, and white matter. We found that infiltrating T-cells were significantly increased in the sulci across all groups compared to controls, with distinct subtypes in RHI without CTE, compared to Low or High CTE. In addition, T-cell infiltration correlated with the duration of RHI, as measured by years of sports play, and synaptic loss. Meningeal and infiltrating T-cells were elevated in sulci with p-tau depositions and spatially related to MHC2 expressing cells. Meningeal T-cells were also significantly correlated with a younger onset of behavioral symptoms. These data suggest that T-cells may play a role in the chronic inflammation and degeneration associated with RHI and CTE.
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