Evidence map›Paper›PMID 41947943›Full record

ReviewImmunoTargets and therapy2026

Guillain-Barré Syndrome: Progress in Diagnosis, Biomarkers, Neuroimaging and Management.

Joumana Freiha, Young Gi Min, Chinar Osman, Yusuf A Rajabally

Abstract readReview
In one paragraph

Review in ImmunoTargets and therapy, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Joumana Freiha *Wessex Neurological Centre, University Hospital Southampton, Southampton, UK.
Young Gi Min *Department of Neurology, Seoul National University College of Medicine, Seoul, Republic of Korea.
Chinar OsmanWessex Neurological Centre, University Hospital Southampton, Southampton, UK.
Yusuf A RajaballyAston Medical School, Aston University, Birmingham, UK.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Guillain-Barré syndrome (GBS) is the leading cause of acute flaccid paralysis worldwide and represents a heterogenous spectrum of acute autoimmune polyradiculoneuropathies. Over the past decade, large international cohorts and advances in neuroimmunology have improved understanding of its clinical subtypes, infectious triggers, and pathophysiology. This review summarises recent progress across key aspects of diagnosis, prognostication, and treatment. We first describe updated perspectives of clinical heterogeneity including GBS variants and geographical variability, and the recognition of autoimmune nodopathies as an important differential diagnosis. We then discuss updated knowledge on antecedent infections including

Indexed as

diagnosisGuillain-Barré syndromepathophysiologyprognosistreatment

Identifiers

PMID41947943
PMCPMC13052254

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.