ReviewJournal of inflammation research2026
Immune Mechanisms in Myocardial Remodeling Following Myocardial Infarction: Focusing on Regulatory Networks of T Cell Responses.
Review in Journal of inflammation research, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
1 citing paper in PubMed.
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Authors and funding
8 authors.
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No grant is acknowledged in the PubMed record.
Abstract
The pathological course of myocardial infarction (MI) entails a complex interplay between innate and adaptive immunity. Although innate immunity plays a dominant role in the tissue damage and clearance during the acute phase, recent studies have shown that T cells also play an important role in myocardial injury and remodeling. Advances in detection technologies have enabled the identification of several autoantigens and clarified the mechanistic roles of distinct T-cell subsets (such as regulatory T cells, helper T cells, cytotoxic T cells, and γδ T cells) during the remodeling phase. These T cell subsets are widely involved in myocardial inflammation, apoptosis and myocardial fibrosis, and play an important role in myocardial remodeling. This review synthesizes current progress on the repertoire of autoantigens recognized after MI, the mechanistic underpinnings of T-cell responses in myocardial remodeling, and emerging immunotherapeutic strategies aimed at mitigating adverse remodeling. Together, these insights provide a conceptual framework to guide future research and therapeutic development in this field.
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