ArticleNeurobiology of language (Cambridge, Mass.)2026
Object Naming After Thalamic Damage: Evidence From a Large-Scale, Chronic-Phase Study of Left Hemisphere Stroke Survivors.
Article in Neurobiology of language (Cambridge, Mass.), 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
Functional imaging and clinical cases implicate the left thalamus in object naming, yet the prevalence of naming impairment after focal thalamic damage is low with variable impact and often rapid resolution. This suggests that compensatory mechanisms, within or beyond the thalamus, may support recovery. We hypothesized that thalamic damage would (a) not cause chronic anomia if other naming-related regions remain intact but (b) exacerbate anomia when co-occurring with damage to non-thalamic naming regions. To test these hypotheses, we retrospectively assessed naming ability in 550 left hemisphere chronic stroke survivors (52% with anomia). Lesion sites included focal thalamic lesions (
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