Evidence map›Paper›PMID 41917398›Full record

ReviewAdvances in experimental medicine and biology2026

Sulfated Glycosaminoglycans in Inflammation.

Satomi Nadanaka, Hiroshi Kitagawa

Abstract readReview
PubMed Publisher
In one paragraph

Review in Advances in experimental medicine and biology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Satomi NadanakaLaboratory of Biochemistry, Kobe Pharmaceutical University, Kobe, Japan.
Hiroshi KitagawaLaboratory of Biochemistry, Kobe Pharmaceutical University, Kobe, Japan. kitagawa@kobepharma-u.ac.jp.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Inflammation has long been regarded as a tissue repair mechanism activated by the body in response to infection or tissue injury. In recent years, chronic inflammation has been implicated as a proinflammatory factor not only in various diseases, such as cancer, atherosclerosis, obesity, and Alzheimer's disease, which increase with age, but also in the aging process itself. What mechanisms cause the inflammatory response that normally dissipates, persists, and becomes chronic? Elucidating the factors that cause chronic inflammation and the mechanisms that induce it will provide insights into the prevention and control of various age-related diseases. In this review, we focus on proteoglycans as factors that cause chronic inflammation and discuss proteoglycans as DAMPs that cause inflammation.

Indexed as

GlycosaminoglycansInflammationInflammation MediatorsAgingAnimalsHumansProteoglycansGlycosaminoglycansInflammation MediatorsProteoglycansChondroitin sulfateDAMPsGlycosaminoglycanHeparan sulfateInflammationProteoglycan

Identifiers

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.