ArticleNPJ precision oncology2026
Targeting histone deacetylation, cell cycle regulators and heat shock proteins as novel therapeutic strategies for penile cancers.
Article in NPJ precision oncology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
Penile carcinoma (PeCa) is a rare malignancy occurring in men. Treatment typically involves penectomy, radiation, or cisplatin-based chemotherapy. A paclitaxel, ifosfamide, and cisplatin-based regimen achieves response rates around 65%. However, non-responders face a 5-year survival rate of only 8%, and due to its rarity, it is mediocrely characterized. Thus, an in-depth delineation of PeCa is crucial to identify novel therapeutic targets. Newly established primary human PeCa cell lines and corresponding xenografts tumors were examined using mass spectrometry and phospho-kinase arrays to analyze the (phospho-)proteome and secretome. Proteomic analyses identified heat shock proteins (HSP27/60/70), factors involved in posttranslational modifications (e.g. acetylation), and the VEGF signaling pathway as putative therapeutic targets. Secreted factors were associated with the HIF-1 and Hippo signaling cascades. Compared to standard chemotherapy (e.g. cisplatin, 5-FU, ifosfamide, irinotecan), treatment with romidepsin, quisinostat (HDAC inhibitors (i)), palbociclib (CDK4/6i), or 17-AAG and PU-H71 (HSP90i) reduced cell viability, induced apoptosis, and led to G2 / M cell cycle arrest in most PeCa cells. This research underscores the therapeutic potential of using HDAC, CDK4/6, and HSP90 inhibitors for PeCa management and reveals additional promising targets and biomarkers for future strategies.
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