ArticleCureus2026
Autoimmune Encephalitis in Later Life: A Missed Opportunity in Routine Geriatric Care.
Article in Cureus, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Authors and funding
5 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Autoimmune encephalitis (AE) is an increasingly recognized but frequently overlooked cause of subacute cognitive and behavioral decline in older adults, in whom delirium is typically attributed to metabolic, infectious, or vascular disorders. We describe an elderly man who presented with persistent encephalopathy following correction of hyponatremia. Despite normal structural neuroimaging and negative cerebrospinal fluid autoimmune markers, he remained delirious. Electroencephalography revealed diffuse slowing, and fluorodeoxyglucose positron emission tomography demonstrated characteristic hypermetabolic activity involving the temporal lobes, basal ganglia, thalami, and cerebellum, strongly suggestive of AE. Initiation of high-dose corticosteroids followed by therapeutic plasma exchange resulted in marked clinical improvement. This case highlights the diagnostic complexities of AE in geriatric practice, particularly the limitations of conventional testing and the value of clinical suspicion backed by functional neuroimaging. Greater clinical vigilance is required to avoid delayed diagnosis and treatment, which may substantially improve outcomes even in severe presentations.
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