ArticleThe Journal of international medical research2026
Involvement of increased arginase-1
Article in The Journal of international medical research, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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13 authors.
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Abstract
BackgroundEndometrial carcinoma is a prevalent gynecologic malignancy with a relatively low survival rate. Emerging studies have demonstrated that crosstalk between group 2 innate lymphoid cells (ILC2s) and myeloid-derived suppressor cells drives tumor progression. The immunopathological mechanisms underlying endometrial carcinoma are not yet fully understood.MethodsThis study assessed the frequency of innate lymphoid cells and myeloid-derived suppressor cells as well as related inflammatory mediators in peripheral blood, carcinoma tissue, and para-cancerous tissue of patients with endometrial carcinoma.ResultsPatients with endometrial carcinoma exhibited decreased levels of interleukin-22 and interferon-γ and increased levels of interleukin-25. Infiltration of ILC2s and monocytic myeloid-derived suppressor cells was elevated, while ILC3 levels were reduced. Functional analysis showed enhanced arginase-1 expression in ILC2s obtained from patients with endometrial carcinoma. Both arginase-1
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