Evidence map›Paper›PMID 41908884›Full record

ArticleExperimental biology and medicine (Maywood, N.J.)2026

Enhanced oxidative stress resilience in

Woori Bae, Mina Norman, Myon Hee Lee

Abstract read
In one paragraph

Article in Experimental biology and medicine (Maywood, N.J.), 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors.

Woori BaeDepartment of Biochemistry and Molecular Pharmacology, New York University School of Medicine, New York, NY, United States.
Mina NormanDepartment of Internal Medicine, Hematology/Oncology Division, Brody School of Medicine at East Carolina University, Greenville, NC, United States.
Myon Hee LeeDepartment of Internal Medicine, Hematology/Oncology Division, Brody School of Medicine at East Carolina University, Greenville, NC, United States.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Oxidative stress is a primary driver of aging, necessitating robust cellular adaptation mechanisms. While peroxisomal β-oxidation and proteasomal degradation are known to influence stress responses, their functional crosstalk remains elusive. In this study, we show that

Indexed as

Caenorhabditis elegansCaenorhabditis elegans ProteinsCatalaseOxidative StressProteasome Endopeptidase ComplexAnimalsMutationParaquatPeroxisomesCaenorhabditis elegans ProteinsCatalaseParaquatProteasome Endopeptidase Complexcatalase (CTL-3)C. elegans agingoxidative stress adaptationperoxisomal β-oxidationproteasome dysfunction

Identifiers

PMID41908884
PMCPMC13021563

What OpenQuestion holds

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LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.