ReviewInternational journal of molecular medicine2026
Crosstalk between autophagy and ferroptosis in liver diseases (Review).
Review in International journal of molecular medicine, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
1 citing paper in PubMed.
- Liver Fibrosis and Purinergic Signaling: Autocrine-Paracrine Role of ATP in Liver Damage.International journal of molecular sciences · 2026Review
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
10 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Ferroptosis is a novel form of regulated cell death triggered by iron‑dependent accumulation in lipid peroxidation. Multiple intracellular catabolic processes and signaling pathways are implicated in ferroptosis regulation. One is autophagy, which delivers cytoplasmic materials to lysosomes for degradation and is critical for the preservation of cellular homeostasis. The discovered role of autophagy in driving ferroptosis have motivated further explorations of the functional interactions and signal pathways between ferroptosis and autophagy, particularly their crosstalk in the pathogenesis or treatment for various liver diseases, such as drug‑induced liver injury, toxin‑induced liver injury, liver fibrosis and hepatocellular carcinoma. The present review presented an in‑depth overview of research on the crosstalk between autophagy and ferroptosis in diverse liver diseases, including the ones aforementioned. The diverse regulatory mechanisms involved in this process are also analyzed to open a new perspective on the interpretation of liver diseases manifestations and provide potential targets for drug discovery and effective intervene.
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Registered trials
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