ArticleJournal of clinical & experimental immunology2026
Benzoate Drugs for Traumatic Brain Injury.
Article in Journal of clinical & experimental immunology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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2 authors.
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Abstract
Traumatic brain injury (TBI) is an acute disorder with prolonged complexities that affirm to be the leading cause of deaths and disabilities in US. The pathophysiology of TBI comprises of multiple complex processes. An impactful insult results in tissue damage, neuroinflammation, impaired cerebral blood flow, BBB disruption, and demyelination, leading to neurodegeneration and functional impairments. There are no specific treatments for TBI. Recent studies highlight the potential efficacy of the following benzoates for attenuating TBI pathologies in rodents: Sodium benzoate, a metabolite of cinnamon, a widely used food additive and a Food and Drug Administration (FDA)-approved drug for glycine encephalopathyGlyceryl tribenzoate, FDA-approved flavoring element being used in food and food packing productions In this review, we have made a genuine attempt to evaluate the significance of these two benzoates as prospective therapeutic options for TBI.
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