ArticleWorld journal of nephrology2026
Renal tubular acidosis complication of non-steroidal anti-inflammatory drugs induced interstitial nephritis and its complete resolution with steroids: A case report.
Article in World journal of nephrology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
backgroundNon-steroidal anti-inflammatory drugs (NSAIDs) are extensively utilized for their analgesic and anti-inflammatory properties; however, their potential to induce renal adverse events remains a significant clinical concern. NSAID-induced interstitial nephritis (IN) and renal tubular acidosis (RTA) are rare but important complications of prolonged NSAID consumption. NSAID-induced RTA has been documented in limited case reports and usually resolves after stopping NSAIDs. However, our case shows a persistent, severe hypokalemia and hypophosphatemia linked to RTA from NSAID-induced IN, which resolved with steroid therapy. CASE SUMMARY: A 70-year-old woman with a history of chronic NSAID use presented with weakness and palpitations. She had atrial fibrillation, severe hypokalemia (K 1.5 mmol/L), non-anion gap metabolic acidosis (HCO
conclusionOur patient exhibited persistent metabolic disturbances even after the cessation of NSAID therapy. Despite moderate chronicity, active interstitial inflammation responded favorably to corticosteroid treatment, resulting in improved renal function and restoration of tubular function, with normalization of acid-base and electrolyte parameters.
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