ArticleMolecular therapy. Oncology2026
Targeted attack: Harnessing myelin-specific plasmolipin for suppression of neuroblastoma and glioblastoma.
Article in Molecular therapy. Oncology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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12 authors.
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Abstract
There is a growing urgency to develop effective strategies for studying and targeting glioblastoma including, which are crucial for tumor initiation and resistance to therapy. Human-immunodeficiency-virus-type-1-based viral particles have emerged as valuable tools in neurology, aiding in the exploration of neural development and tumorigenesis, with promising clinical applications on the horizon. We targeted proteolipid plasmolipin, found on glial cell surfaces, acting as an entry receptor for the McERV endogenous murine retrovirus, with replication-defective vectors carrying either a fluorescent marker or the suicide gene TK.007, pseudotyped with the McERV envelope protein. We evaluated these vectors' efficiency in delivering genes to glioblastoma and neuronal cells both
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