Evidence map›Paper›PMID 41880088›Full record

ReviewMolecular biology reports2026

MYC: unveiling novel therapeutic avenues for endometriosis.

Xiaofang Ge, Chenjing Yue, Maoxiang Zhang, Mengze Wang, Jiayu Liu, Aifang Jiang, Zhenhai Yu

Abstract readReview
PubMed Publisher
In one paragraph

Review in Molecular biology reports, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Xiaofang Ge *Department of Reproductive Medicine, Affiliated Hospital of Shandong Second Medical University, Weifang, Shandong Province, P.R. China.
Chenjing Yue *Department of Reproductive Medicine, Affiliated Hospital of Shandong Second Medical University, Weifang, Shandong Province, P.R. China.
Maoxiang ZhangDepartment of Reproductive Medicine, Affiliated Hospital of Shandong Second Medical University, Weifang, Shandong Province, P.R. China.
Mengze WangDepartment of Reproductive Medicine, Affiliated Hospital of Shandong Second Medical University, Weifang, Shandong Province, P.R. China.
Jiayu LiuDepartment of Reproductive Medicine, Affiliated Hospital of Shandong Second Medical University, Weifang, Shandong Province, P.R. China.
Aifang JiangDepartment of Reproductive Medicine, Affiliated Hospital of Shandong Second Medical University, Weifang, Shandong Province, P.R. China. aifangjiang@163.com.
Zhenhai YuDepartment of Reproductive Medicine, Affiliated Hospital of Shandong Second Medical University, Weifang, Shandong Province, P.R. China. tomsyu@163.com.

Funding

National Natural Science Foundation of China 81972489
6 · The paper itself

Abstract

The MYC family has been the subject of extensive research in the field of cancer, yet the mechanisms by which these factors operate in the context of endometriosis remain unclear. Considering the existing studies on the MYC family and diseases of the female reproductive system, this review examines the role of MYC family members in regulating cell proliferation and metabolic activity in endometriosis. Furthermore, the study explores the potential association between the MYC family and endometriosis, drawing parallels with its established role in cancer and findings from other gynecological diseases. The primary areas of discussion include cell proliferation and invasion, hormone dependence, and epigenetic regulation. The review also considers inhibitors of the MYC family and other related molecules that may affect MYC expression. The findings of this study suggest significant potential for the MYC family to play a pivotal role in the context of endometriosis. It is anticipated that advancements in technology will stimulate further research in this area and facilitate the development of clinically applicable therapeutic strategies.

Indexed as

EndometriosisProto-Oncogene Proteins c-mycAnimalsCell ProliferationEpigenesis, GeneticFemaleHumansMYC protein, humanProto-Oncogene Proteins c-mycEndometriosisEpigeneticHormoneMYCTreatment

Identifiers

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.