Evidence map›Paper›PMID 41849049›Full record

ReviewMolecular neurobiology2026

CaMKII as a Multimodal Signalling Hub in Neural Connectivity and Vulnerability.

Stephanie Olliff, Vinod Sundaramoorthy

Abstract readReview
PubMed Full text at PMC Publisher
In one paragraph

Review in Molecular neurobiology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Stephanie OlliffCommonwealth Scientific and Industrial Research Organisation (CSIRO), Australian Centre for Disease Preparedness (ACDP), East Geelong, VIC, Australia.
Vinod SundaramoorthyCommonwealth Scientific and Industrial Research Organisation (CSIRO), Australian Centre for Disease Preparedness (ACDP), East Geelong, VIC, Australia. vinod.sundaramoorthy@csiro.au.

Funding

Australian Research Council DE210101145
6 · The paper itself

Abstract

Calcium/calmodulin-dependent protein kinase II (CaMKII) is one of the most abundant and versatile signalling molecules in the brain, uniquely positioned to convert transient signals into durable structural and functional changes. Classical models cast CaMKII as a Ca

Indexed as

Calcium-Calmodulin-Dependent Protein Kinase Type 2NeuronsSignal TransductionAnimalsHumansNeuronal PlasticityCalcium-Calmodulin-Dependent Protein Kinase Type 2Axon growth and degenerationCaMKII isoform diversityCaMKII signallingKinase–phosphatase balanceProtein phosphataseSynaptic plasticity

Identifiers

PMID41849049
PMCPMC12999712

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.