ArticleSensors (Basel, Switzerland)2026
A Study on Autonomous Driving Motion Sickness from the Perspective of Multimodal Human Signals.
Article in Sensors (Basel, Switzerland), 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
1 citing paper in PubMed.
- Quantifying the Relative Contributions of Exposure Time, ECG, GSR, and Vibration-Derived Features for Motion Sickness Prediction.Sensors (Basel, Switzerland) · 2026Article
Corrections and comments
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Authors and funding
2 authors.
Funding
Abstract
In autonomous driving, motion sickness (MS) arises from physical or visual stimuli, or a combination of both. However, objective quantification of MS level (MSL) remains limited beyond questionnaire-based assessments. Using multimodal human signals (physiological and behavioral) collected in an autonomous driving simulator, this study addresses the association between these signals and MSL, across these MS types, by (i) screening and curating a decade of human-signal MS studies (HS-Set) to establish a data-driven foundation for selecting target sensor domains and features, (ii) constructing a dataset with subjective measures of MSL (fast motion sickness scale and simulator sickness questionnaire (SSQ)), alongside human signals (electroencephalogram (EEG), photoplethysmogram (PPG), electrodermal activity (EDA), skin temperature, and head/eye movement), (iii) conducting a correlation analysis between MSL and the identified features from HS-Set, and (iv) quantifying multivariable contributions at the feature and sensor domains through an explainable boosting machine (EBM). Key correlations include head amplitude/energy (pitch/surge) with SSQ total/oculomotor, eye entropy with nausea/oculomotor (positive), and EDA with nausea (negative). The EBM-based contribution analysis highlights EEG connectivity and head kinematics as dominant contributors; excluding EEG, the interpretability of single-domain models remains limited. Additionally, a combination of Head, PPG, and EDA domains retains over 80% of the full model's interpretability.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.