ArticleJournal of nanobiotechnology2026
Neutrophil membrane-coated and MMP2-responsive nanoparticles deliver PIM1 inhibitor to alleviate inflammatory arthritis through inhibiting Th17 cell differentiation.
Article in Journal of nanobiotechnology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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12 authors.
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Abstract
backgroundInflammatory arthritis (IA) is a group of chronic progressive inflammatory diseases characterized by the destruction of joints. The clinical efficacy and safety of the current drugs for patients with IA still need improvement, suggesting the importance of developing new therapeutic agents with the potential to specifically target inflammatory sites and precisely intervene in pathogenic molecules.
resultsPreviously, we demonstrated that elevated PIM1 expression in CD4
conclusionThese results suggested that NM@MRP-NP was a structurally defined, biologically stable, inflammatory-targeted and conditionally releasing nanotherapeutic system with efficient drug delivery, which could provide new insight into the targeted interventions for IA.
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