ArticleActa histochemica et cytochemica2026
Why Does DNA Methylation Accumulate at CpG Loci?: Sequential Analysis of
Article in Acta histochemica et cytochemica, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
1 citing paper in PubMed.
- The role of methylation in pituitary neuroendocrine tumors current insights and emerging perspectives.Molecular biology reports · 2026Review
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Authors and funding
1 author.
Funding
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Abstract
DNA methylation is a key epigenetic modification that regulates transcriptional activity and is frequently altered during carcinogenesis. To elucidate how cytosine methylation becomes selectively localized to CpG loci during tumor development, we analyzed sequential renal lesions induced by streptozotocin (STZ) in rats. Using microdissection combined with an agarose-bead-based bisulfite sequencing method, the methylation status of the
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Registered trials
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