ReviewExploration (Beijing, China)2026
Scar Inhibition in Wound Healing: Mechanisms, Design, and Recent Advances.
Review in Exploration (Beijing, China), 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
5 citing papers in PubMed.
- NIR-II Fluorescent Nanoplatforms with Defect-Regulated Piezoelectricity for Dual NIR-II/MRI-Guided, Hypoxia-Resilient Piezo-Chemodynamic Therapy and cGAS-STING Activation in Orthotopic Liver Tumor.Small (Weinheim an der Bergstrasse, Germany) · 2026Article
- Nanotechnology in Dermatology: A Comprehensive Narrative Review of Performance, Safety, and Clinical Translation.Health science reports · 2026Article
- Advanced Hydrogel Functionalization Strategies for Burn-Related Wound Treatment.Biomaterials research · 2026Review
- Electric-based dressing for wound management.Frontiers in cellular and infection microbiology · 2026Review
- A PIEZO-Gated Mechanotransduction Timing Window Governs Scar Commitment in Wound Healing.Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair SocietyReview
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
5 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Scar inhibition plays a crucial role in wound healing, particularly in the prevention of excessive scar formation during skin repair. While scar formation is a natural part of the healing process, improper scarring can lead to functional impairment, cosmetic defects, and psychological impacts. Therefore, understanding the mechanisms behind scar inhibition and exploring therapeutic strategies is essential for improving clinical outcomes. This review summarizes the primary mechanisms of scar formation and inhibition, including the regulation of collagen deposition, modulation of the inflammatory response, and control of cell proliferation and migration. In recent years, novel therapeutic approaches have emerged for scar inhibition, including gene therapy, stem cell treatments, and localized drug delivery systems, and the use of biomaterials. These methods not only enhance the effectiveness of scar treatment but also improve the biocompatibility and durability of the healing process. Although some of these approaches have shown promising results in early-stage studies, challenges remain for clinical applications, such as the individualization of treatment plans and the sustainability of outcomes. Finally, this review discusses future research directions and proposes strategies to enhance the potential of scar inhibition therapies and their clinical translation.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.